Personal Research Database Bibliometric

Full Text: 2014\Tum Biol35, 8445.pdf

Abstract: A number of studies have reported that HOTAIR expression levels were higher in cancerous tissues than in corresponding noncancerous tissues and overexpression of HOTAIR was prone to lymph node metastasis. This meta-analysis collected all relevant articles and explored the association between HOTAIR expression levels with lymph node metastasis. A literature collection was conducted by searching electronic databases PubMed, Cochrane Library, OVID, Web of Science, and CNKI (up to March 22, 2014). The odds ratio (OR) and its corresponding 95 % confidence interval (CI) were calculated to assess the strength of the association by using RevMan5.2 software. A total of 748 patients from 8 studies were included in this meta-analysis. The results showed there was a significant difference in the incidence of lymph node metastasis between high HOTAIR expression group and low HOTAIR expression group (OR = 2.81, 95 % CI 1.38-5.70, P = 0.004 random-effects model). This meta-analysis demonstrated that the incidence of lymph node metastasis in patients detected with high HOTAIR expression was higher than that in patients with low HOTAIR expression.

Keywords: Articles, Association, Cancer, Cancer, Carcinoma Progression, Collection, Confidence, Databases, Expression, From, Hotair, Incidence, Interval, Literature, Lncrna, Lymph Node Metastasis, Marker, Meta Analysis, Meta-Analysis, Metaanalysis, Metastasis, Model, Odds Ratio, P, Patients, Poor-Prognosis, Pubmed, Random Effects Model, Rna, Science, Software, Strength, Survival, Web Of Science

? Xie, Z.B., Ma, L., Wang, X.B., Bai, T., Ye, J.Z., Zhong, J.H. and Li, L.Q. (2014), Transarterial embolization with or without chemotherapy for advanced hepatocellular carcinoma: A systematic review. Tumor Biology, 35 (9), 8451-8459.

Full Text: 2014\Tum Biol35, 8451.pdf

Abstract: Transarterial chemoembolization (TACE) and transarterial embolization (TAE) are commonly used as first-line treatment for patients with advanced hepatocellular carcinoma (HCC) and have been shown to improve overall survival (OS). However, there remain concerns regarding whether the benefit of the prolonged survival achieved with TACE is superior to the maximum cytotoxic effect of the associated chemotherapeutics. This systematic review aims to compare the efficiency of TACE and TAE based on randomized controlled trials (RCTs). MEDLINE, EMBASE, the Cochrane library, the Science Citation Index, and the Chinese National Knowledge Infrastructure databases were systematically searched through the end of April 2014. Risk ratios (RRs) and 95 % confidence intervals (CIs) were calculated. Meta-analysis of the RCTs was conducted to estimate the mortality and survival rate between the TACE and TAE groups. The analysis included five RCTs involving 582 patients. For all-cause mortality, TACE did not result in a statistically significant reduced incidence of adverse events than TAE with a pooled RR of 1.21 (95 % CI = 0.74-1.98, P = 0.16). In addition, 6-, 9-, 12-, 24-, and 36-month OS of the TACE group were not significantly higher than that of the TAE group (all P > 0.05). Interestingly, TACE resulted in a significantly higher rate of advanced events. The efficacy of TACE is not superior to TAE in advanced HCC patients. Moreover, TACE was associated with an increased rate of adverse events than TAE. Improved strategies are needed to reduce the risk of post-TACE complications.

Keywords: Advanced Hepatocellular Carcinoma, Adverse Events, Analysis, Carcinoma, Chemoembolization, Chemotherapy, Chinese, Citation, Complications, Confidence, Confidence Intervals, Cytotoxic, Databases, Drug, Efficacy, Efficiency, Embase, Embolization, Events, First Line, Groups, Guidelines, Hcc, Hepatocellular Carcinoma, Incidence, Intervals, Knowledge, Management, Medline, Meta Analysis, Meta-Analysis, Metaanalysis, Mortality, Overall Survival, P, Patients, Prolonged, Randomized, Randomized Controlled Trials, Randomized Controlled-Trials, Review, Risk, Science, Science Citation Index, Solid Tumors, Survival, Survival Rate, Survivin, Systematic, Systematic Review, Tace, Therapy Options, Transarterial Chemoembolization, Transarterial Embolization, Transcatheter Arterial Embolization, Treatment

? Wei, K.K., Jiang, L., Wei, Y.Y., Wang, Y.F., Qian, X.K., Dai, Q. and Guan, Q.L. (2014), The prognostic value of ERCC1 expression in gastric cancer patients treated with platinum-based chemotherapy: A meta-analysis. Tumor Biology, 35 (9), 8721-8731.

Full Text: 2014\Tum Biol35, 8721.pdf

Abstract: Numerous studies examined the association between excision repair complementation group 1 (ERCC1) expression and the prognosis of gastric cancer patients receiving platinum-based chemotherapy but yielded controversial results. We thus conducted a meta-analysis to quantitatively evaluate the prognostic value of ERCC1 expression in gastric cancer patients receiving platinum-based chemotherapy. A systematic literature search was performed to identify relevant studies in PubMed, Web of Science, Embase, Cochrane Library, China National Knowledge Infrastructure, Chinese Biomedical Literature Database, and WanFang Database up to December 17, 2013. Pooled hazard ratios (HRs) or odds ratios (ORs) with 95 % confidence intervals (CIs) were estimated. Moreover, meta-regression analysis and subgroup analysis were conducted according to ethnicity, HR extraction, detection methods, survival analysis, and quality score. A total of 1,409 patients from 21 studies were subjected to final analysis. Positive/high ERCC1 expression was significantly associated with poorer overall survival (HR, 1.58; 95 % CI, 1.09-2.28), especially in Asians (HR, 1.81; 95 % CI, 1.20-2.73), and lower response rate (OR, 0.26; 95 % CI, 0.18-0.36), but not with clinicopathological features, such as gender (OR, 1.01; 95 % CI, 0.68-1.51), grade (OR, 0.66; 95 % CI, 0.43-1.01), and stage (OR, 1.05; 95 % CI, 0.58-1.90). This meta-analysis suggested that ERCC1 expression might be a useful biomarker to predict response and survival for gastric cancer patients receiving platinum-based chemotherapy, particularly in Asians.

Keywords: Adjuvant Chemotherapy, Analysis, Asians, Association, Biomarker, Biomedical, Cancer, Chemotherapy, China, Chinese, Cisplatin-Based Chemotherapy, Confidence, Confidence Intervals, Database, Dna-Repair, Ercc1, Ethnicity, Expression, Extraction, From, Funnel Plots, Gastric, Gastric Cancer, Gastroesophageal Cancer, Gender, Hazard, Intervals, Knowledge, Literature, Literature Search, Lung-Cancer, Messenger-RNA Levels, Meta Analysis, Meta-Analysis, Meta-Regression, Metaanalysis, Methods, Overall Survival, Patients, Platinum-Based Chemotherapy, Prognosis, Prognostic, Protein Expression, Publication Bias, Pubmed, Quality, Repair, Response, Science, Survival, Survival Analysis, Systematic, Systematic Literature Search, Thymidylate Synthase, Value, Web Of Science

? Wang, X., Zhu, Y.B., Cui, H.P. and Yu, T.T. (2014), Aberrant promoter methylation of p15^INK4b and p16^INK4a genes may contribute to the pathogenesis of multiple myeloma: A meta-analysis. Tumor Biology, 35 (9), 9035-9043.

Full Text: 2014\Tum Biol35, 9035.pdf

Abstract: We carried out the current meta-analysis aiming to comprehensively assess the potential role of p15 (INK4b) and p16 (INK4a) aberrant promoter methylation in the pathogenesis of multiple myeloma (MM). The MEDLINE (1966 2013), Cochrane Library (Issue 12, 2013), EMBASE (1980 2013), CINAHL (1982 2013), Web of Science (1945 2013), and Chinese Biomedical (CBM) (1982 2013) databases were searched without language restrictions. Meta-analyses were conducted using Stata software (Version 12.0, Stata Corporation, College Station, TX, USA). Odds ratios (ORs) and their 95 % confidence intervals (95 %CIs) were calculated. Thirteen clinical case-control studies, which enrolled a total of 465 MM patients and 180 healthy subjects, were included in the meta-analysis. The results of our meta-analysis demonstrated that the frequencies of p15 (INK4b) and p16 (INK4a) promoter methylation in cancer samples were significantly higher than in normal samples (p15 (INK4b) : OR = 6.26, 95 %CI = 3.87 10.12, P < 0.001; p16 (INK4a) : OR = 2.26, 95 %CI = 1.22 4.20, P < 0.001). Ethnicity-stratified analysis showed that the aberrant methylation of p15 (INK4b) was significantly related with the risk of MM among both Caucasians and Asians (all P < 0.05). Furthermore, our results also illustrated a strong positive correlation between p16 (INK4a) promoter methylation and the pathogenesis of MM among Asians (OR = 5.17, 95 %CI = 3.45 7.74, P < 0.001), but not among Caucasians (P > 0.05). The current meta-analysis confirms and reinforces existing findings that p15 (INK4b) and p16 (INK4a) promoter methylation may be closely implicated in the pathogenesis of MM.

Keywords: Analysis, Asians, Biomedical, Cancer, Case-Control, Case-Control Studies, Chinese, Clinical, Confidence, Confidence Intervals, Correlation, Databases, Embase, Genes, Inactivation, Intervals, Language, Medline, Meta Analysis, Meta-Analyses, Meta-Analysis, Metaanalysis, Methylation, Multiple Myeloma, Normal, P, P14(Arf), P15, P15(Ink4b), P15(Ink4b), P16, P16(Ink4a), P16(Ink4a), Pathogenesis, Patients, Potential, Restrictions, Risk, Role, Science, Software, Squamous-Cell Carcinoma, Stata, Tumor-Suppressor Genes, USA, Web Of Science

? Yao, F., Yan, S.S., Wang, X.C., Shi, D.H., Bai, J.Y., Li, F., Sun, B.C. and Qian, B. (2014), Role of IL-17F T7488C polymorphism in carcinogenesis: A meta-analysis. Tumor Biology, 35 (9), 9061-9068.

Full Text: 2014\Tum Biol35, 9061.pdf

Abstract: Previous case-control studies on the association of interleukin-17F (IL-17F) T7488C polymorphism and cancer risk have yielded conflicting and inconclusive findings. We performed a meta-analysis by pooling all currently available data to acquire a more precise estimation of the association. A comprehensive literature screening from the PubMed, Embase, Web of Science, and Wanfang databases was performed for eligible publications without language restrictions. The pooled odds ratios (ORs) with corresponding 95 % confidence intervals (95 % CIs) were calculated. According to the inclusion criteria, a total of nine case-control studies with 3,034 cases and 3,694 controls were included. Overall, the pooled ORs showed that IL-17F T7488C polymorphism was associated with neither increased nor decreased risk of cancer. However, the IL-17F T7488C polymorphism exerted risk effect on cancer in population-based case-control studies when stratifying analysis by source of controls (C vs T OR = 1.24, 95 % CI, 1.10-1.40, pooled OR (P-OR) < 0.001; TC vs TT OR = 1.28, 95 % CI, 1.11-1.48, P-OR = 0.001; CC + TC vs TT OR = 1.29, 95 % CI, 1.12-1.48, P-OR < 0.001). Additionally, the variant genotypes of IL-17F T7488C could alter the risk of gastric cancer under the following comparisons (C vs T OR = 1.29, 95 % CI, 1.13-1.47, P-OR < 0.001; TC vs TT OR = 1.35, 95 % CI, 1.14-1.60, P-OR < 0.001; CC + TC vs TT OR = 1.35, 95 % CI, 1.15-1.58, P-OR < 0.001). Sensitivity analysis by sequential omission of single study did not materially alter the pooled findings. The present meta-analysis suggests that the IL-17F T7488C polymorphism may modify the risk of cancer, particularly gastric cancer. However, the precise association needs to be elucidated by more individual studies with sufficient statistical power.

Keywords: Analysis, Association, Cancer, Cancer Risk, Case-Control, Case-Control Studies, Cells, Colorectal-Cancer, Confidence, Confidence Intervals, Criteria, Data, Databases, Diseases, Family, From, Gastric, Gastric Cancer, Gastric-Cancer, Graphical Test, Growth, Inflammation, Interleukin-17a And-17f Genes, Interleukin-17f, Intervals, Language, Literature, Meta Analysis, Meta-Analysis, Metaanalysis, Needs, Polymorphism, Population Based, Population-Based, Power, Publications, Pubmed, Restrictions, Risk, Science, Screening, Sensitivity, Sensitivity Analysis, Sequential, Source, Statistical Power, Web Of Science

? Sui, L., Liu, K., Shen, W. and Zhang, L. (2014), Relationships between VEGF protein expression and pathological characteristics of diffuse large B cell lymphoma: A meta-analysis. Tumor Biology, 35 (9), 9085-9093.

Full Text: 2014\Tum Biol35, 9085.pdf

Abstract: We carried out the current meta-analysis of relevant cohort studies in an attempt to investigate the relationships between vascular endothelial growth factor (VEGF) expression and pathological characteristics of diffuse large B cell lymphoma (DLBCL). The following electronic databases were searched for relevant articles without any language restrictions: Web of Science (1945 2013), the Cochrane Library Database (Issue 12, 2013), PubMed (1966 2013), EMBASE (1980 2013), CINAHL (1982 2013), and the Chinese Biomedical Database (CBM) (1982 2013). Meta-analyses were conducted with the use of STATA software (version 12.0, Stata Corporation, College Station, TX, USA). Odds ratios (ORs) and its 95 % confidence interval (95 % CI) were calculated. Nine clinical cohort studies with a total of 789 DLBCL patients met our inclusion criteria. The meta-analysis results showed that patients with positive VEGF expression had higher international prognostic index (IPI) scores than VEGF-negative patients (OR = 5.12, 95 % CI = 2.70 9.71, P < 0.001). There was a significantly positive association between positive VEGF expression and evaluated lactate dehydrogenase (LDH) levels (OR = 2.50, 95 % CI = 1.36 4.60, P = 0.003). We also found that patients with positive B symptoms had increased level of VEGF expression (OR = 2.02, 95 % CI = 1.08 3.77, P = 0.027). The findings of our meta-analysis provide reliable evidence that VEGF expression may be strongly correlated with pathological characteristics of DLBCL.

Keywords: Angiogenesis, Articles, Association, Biomedical, Cancer, Cell, Characteristics, Chinese, Clinical, Cohort, Confidence, Criteria, Database, Databases, Diffuse Large B Cell Lymphoma, Embase, Endothelial Growth-Factor, Evidence, Expression, Growth, Growth Factor, Index, International, Interval, Lactate, Language, Ldh, Lymphoma, Meta Analysis, Meta-Analyses, Meta-Analysis, Metaanalysis, P, Patients, Predict, Prognostic, Protein, Pubmed, Restrictions, Science, Serum, Software, Stata, Survival, Symptoms, Time, USA, Vascular Endothelial Growth Factor, VEGF, Version, Web Of Science

? Chen, Y.Z., Liu, D., Zhao, Y.X., Wang, H.T., Gao, Y. and Chen, Y. (2014), Aberrant promoter methylation of the SFRP1 gene may contribute to colorectal carcinogenesis: A meta-analysis. Tumor Biology, 35 (9), 9201-9210.

Full Text: 2014\Tum Biol35, 9201.pdf

Abstract: This meta-analysis of published cohort studies was conducted to evaluate whether promoter methylation of the secreted frizzled-related protein 1 (SFRP1) gene contributes to colorectal carcinogenesis. The Web of Science (1945 similar to 2013), the Cochrane Library Database (Issue 12, 2013), PubMed (1966 similar to 2013), EMBASE (1980 similar to 2013), CINAHL (1982 similar to 2013), and the Chinese Biomedical Database (CBM) (1982 similar to 2013) were searched without language restrictions. Meta-analysis was conducted using the STATA 12.0 software. We calculated odds ratio (OR) and its 95 % confidence interval (95 % CI) to estimate the correlations between SFRP1 promoter methylation and colorectal carcinogenesis. In the present meta-analysis, 8 cohort studies with a total of 942 patients with colorectal cancer (CRC) were included. The pooled results revealed that the frequency of SFRP1 promoter methylation in cancer tissues were significantly higher than those of normal, adjacent, and benign tissues (cancer tissues vs. normal tissues: OR = 31.49, 95 % CI = 17.57 similar to 56.44, P < 0.001; cancer tissues vs. adjacent tissues: OR = 5.95, 95 % CI 3.12 - 10.00, P < 0.001; cancer tissues vs. benign tissues: OR = 3.01, 95 % CI 1.72 similar to 5.27, P < 0.001; respectively). Furthermore, ethnicity-stratified analysis indicated that SFRP1 promoter methylation was strongly correlated with colorectal carcinogenesis among both Asians and Caucasians (all P < 0.05). Our findings provide empirical evidence that SFRP1 promoter methylation may be correlated with the pathogenesis of CRC.

Keywords: Analysis, Asians, Biomedical, Cancer, Cancer Patients, Cell-Proliferation, Chinese, Cohort, Colorectal Cancer, Colorectal Carcinogenesis, Confidence, Correlations, Database, Disease, Embase, Epidemiology, Evidence, Gene, Interval, Language, Meta Analysis, Meta-Analysis, Metaanalysis, Methylation, Normal, Odds Ratio, P, Pathogenesis, Patients, Promoter Methylation, Protein, Pubmed, Restrictions, Science, Sfrp1, Signaling Pathway, Software, Web Of Science, Wnt Antagonist Sfrp1

? Yin, D., Jiang, Y., Wang, N., Ouyang, L., Lu, Y.M., Zhang, Y., Wei, H. and Zhang, S.L. (2014), The diagnostic value of serum hybrid capture 2 (CH2) HPV DNA in cervical cancer: A systematic review and meta-analysis. Tumor Biology, 35 (9), 9247-9253.

Full Text: 2014\Tum Biol35, 9247.pdf

Abstract: The diagnostic accuracy of cervical cancer remains a clinical challenge, and a number of studies have used the serum hybrid capture 2 (HC2) human papillomavirus (HPV) DNA in the diagnosis of cervical cancer. The aim of the present meta-analysis was to determine the overall accuracy of HC2 HPV DNA in the diagnosis of cervical cancer. A systematic review of studies from PubMed, Embase, the Cochrane Library, Web of Science, Ovid, Chinese Biomedical Literature Database-disc, Chinese National Knowledge Infrastructure (CNKI), Technology of Chongqing (VIP), and Wan Fang database was conducted, and the data concerning the accuracy of HC2 HPV DNA in the diagnosis of cervical cancer were pooled. The methodological quality of each study was assessed by quality assessment for studies of diagnostic accuracy (QUADAS). Statistical analysis was performed by employing Meta-DiSc (version 1.4) and Stata (version 12.0) software. The overall test performance was summarized using receiver operating characteristic curves. Finally, 12 studies, including 12,492 subjects, met the inclusion criteria and then included in this present meta-analysis. The summary estimates for serum HC2 HPV DNA in the diagnosis of cervical cancer were as follows: sensitivity 0.83 (95 % confidence interval (CI) 0.81-0.85), specificity 0.71 (95 % CI 0.69-0.72), positive likelihood ratio 3.65 (95 % CI 1.77-7.54), negative likelihood ratio 0.32 (95 % CI 0.21-0.48), and diagnostic odds ratio 10.54 (95 % CI 4.95-22.46), and the area under the curve was 0.8922. Our findings suggest that HC2 HPV DNA may improve the accuracy of cervical cancer diagnosis, while the results of HC2 HPV DNA assays should be interpreted in parallel with conventional test results and other clinical findings.

Keywords: Accuracy, Analysis, Assessment, Biomedical, Cancer, Cervical Cancer, Challenge, Chinese, Clinical, Clinical Findings, Confidence, Conventional, Criteria, Curve, Cytology, Data, Database, Diagnosis, Diagnostic, Diagnostic Accuracy, Dna, Estimates, From, Hc2hpvdna, Hpv, Human, Human Papillomavirus, Hybrid, Hybrid Capture 2 (Hc2), Interval, Knowledge, Likelihood Ratio, Literature, Meta Analysis, Meta-Analysis, Metaanalysis, Odds Ratio, Papillomavirus, Performance, Pubmed, Quality, Quality Of, Review, Science, Sensitivity, Serum, Smear, Software, Specificity, Stata, Statistical Analysis, Systematic, Systematic Review, Technology, Tests, Value, Version, Web Of Science

? Gu, X., Xue, J.Q., Zhu, X., Ye, M.S. and Zhang, W.H. (2014), Aberrant promoter methylation of the CHD1 gene may contribute to the pathogenesis of breast cancer: A meta-analysis. Tumor Biology, 35 (9), 9395-9404.

Full Text: 2014\Tum Biol35, 9395.pdf

Abstract: Cadherin-1 (CHD1), as an invasion suppressor gene, could suppress tumor cell invasion and metastasis in various tumors, but reduced CHD1 levels, resulting from epigenetic silencing, are common in poorly differentiated, advanced stage carcinomas. This meta-analysis was performed to evaluate the relationships between promoter methylation of CHD1 and breast cancer. Relevant studies were retrieved from the Web of Science (1945 similar to 2013), the Cochrane Library (Issue 12, 2013), PubMed (1966 similar to 2013), EMBASE (1980 similar to 2013), CINAHL (1982 similar to 2013), and the Chinese Biomedical Database (CBM) (1982 similar to 2013) using a systematic literature search. Results were summarized by meta-analyses, conducted using the STATA software (version 12.0, Stata Corporation, College Station, TX, USA). Odds ratios (ORs) and 95 % confidence intervals (95 % CIs) were calculated. In the present meta-analysis, 9 cohort studies with a total of 425 patients with breast cancer were included. Our meta-analysis results demonstrated that the frequency of CHD1 promoter methylation in cancer tissues was significantly higher than that in normal tissues, adjacent tissues, and benign tissues (cancer tissue vs. normal tissue OR = 30.87, 95 % CI = 16.76 similar to 56.86, P < 0.001; cancer tissue vs. adjacent tissue OR = 23.30, 95 % CI = 12.85 similar to 42.26, P < 0.001; cancer tissue vs. benign tissue OR = 2.94, 95 % CI = 1.60 similar to 5.40, P < 0.001; respectively). Ethnicity-stratified analysis indicated that aberrant CHD1 promoter methylation was strongly correlated with breast cancer among both Asians and Caucasians in the majority of subgroups. Our results suggest that aberrant promoter methylation of the CHD1 gene may have a high frequency in breast cancer tissues. Thus, CHD1 methylation could be correlated with the pathogenesis of breast cancer.

Keywords: Analysis, Asians, Biomedical, Breast Cancer, Cancer, Cdh1, Cell, Chd1, Chinese, Cohort, Confidence, Confidence Intervals, Database, E-Cadherin Expression, Embase, From, Gene, Hypermethylation, Intervals, Literature, Literature Search, Meta Analysis, Meta-Analysis, Metaanalysis, Metastasis, Methylation, Normal, P, Pathogenesis, Patients, Promoter Methylation, Pubmed, Results, Science, Software, Stata, Statistics, Systematic, Systematic Literature Search, Tissue, Tumor, USA, Version, Web Of Science, Women

? Chen, Y., Yu, Z.G., Zhang, B., Chang, Z.Q., Wang, H. and Liu, Z.D. (2014), CRR9p polymorphism as a protective factor for lung cancer. Tumor Biology, 35 (10), 9557-9562.

Full Text: 2014\Tum Biol35, 9557.pdf

Abstract: A number of studies have investigated the association between CRR9p polymorphism and risk of lung cancer (LC), yet the role in LC pathogenesis remains unclear owing to inconsistencies across studies. We searched PubMed, Embase, and Web of Science for all medical literature published until January 2014. Pooled odds ratios (ORs) and 95 % confidence intervals (CIs) were obtained by means of the fixed effects model. Data from eight studies satisfying the predesigned inclusion criteria were selected for this meta-analysis. We found a statistically significant evidence for a protective effect on the overall LC risk (TT vs. CC: OR=0.78, 95 % CI=0.70-0.87, P-het=0.299; TT vs. CT+CC: OR=0.81, 95 % CI=0.73-0.90, P-het=0.113; T vs. C: OR=0.90, 95 % CI=0.86-0.95, P-het=0.758; TT+CT vs. CC: OR=0.92, 95 % CI=0.87-0.98, P-het=0.892). Both Caucasian and Asian populations were suggested to have a reduced risk of developing such cancer. In the analysis of the association between rs401681 and non-small cell lung cancer (NSCLC) risks, all of the contrast models showed similar results except the CT vs. CC genetic model (OR=0.93, 95 % CI=0.84-1.02, P-het= 0.568). Our meta-analysis provides supportive evidence that CRR9p polymorphism may influence a risk of LC and NSCLC in a protective model.

Keywords: Adenocarcinoma, Analysis, Asian, Association, Cancer, Caucasian, Cell, Confidence, Confidence Intervals, Criteria, CRR9P, CT, Data, Developing, Effects, Evidence, Fixed Effects Model, From, Gene Polymorphisms, Genetic, Genome-Wide Association, Influence, Intervals, Korean Population, Literature, Lung, Lung Cancer, Medical, Medical Literature, Meta Analysis, Meta-Analysis, Metaanalysis, Model, Models, Mortality, NSCLC, Pathogenesis, Polymorphism, Populations, Pubmed, Risk, Risks, Role, Science, Susceptibility Locus, Telomere Length, Variants, Web, Web Of Science

? Feng, B., Fan, Y.G., Wang, W., Yao, G.L. and Zhai, J.M. (2014), IL-17A G197A and C1249T polymorphisms in gastric carcinogenesis. Tumor Biology,